Spinal cord injury is a serious disease that threatens human health. Preventive treatment and rehabilitation of spinal cord injury has become a major research topic in the medical community today. Secondary injury occurs after spinal cord injury, involving many aspects such as circulation, edema, neurotransmitters, ion changes, nitric oxide, vascular endothelin, and apoptosis. Using non-damage micro-measurement technology, it was found that the current and voltage of ions play a major role in regulating secondary damage, especially the balance of Ca2 + flow (internal and external flow).

Scientist Porterfield of Purdue University and others used non-invasive micro-measurement technology to study the changes of strong biological currents and Ca2 + in guinea pig spinal cord trauma into cells and tissues. It was found that a strong biological current traverses the spinal cord trauma, this endogenous current decreases with time and away from the trauma, and eventually maintains a low and stable intensity, while detecting a significant calcium influx. The results of the study suggest that current and Ca2 + are related to the death, distortion, and asymmetry of organelles in damaged nerve fibers. These data increase our understanding of the mechanism of secondary injury and provide a new direction to consider how to prevent tissue alienation and continuous damage.

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